Spending for DVDs and Blu-ray discs is falling so fast that 2012 likely will be first year when consumers will pay more to buy and rent movies via the Internet, according to a report today from IHS Screen Digest Video Intelligence Service. The research firm says that the average household will spend $123.50 to buy and rent discs this year ? down from $133.21 last year and $149.53 in 2010. The average home spent $206.78 in 2006, when Blu-ray discs were introduced. Looked at another way, people spent $8.8B last year to buy packaged videos, down 12% vs 2010. That will keep falling to $5.4B in 2016 ? which IHS notes is ?the level in 1997 when DVDs were first launched.? Meanwhile, consumers paid $5.7B to rent discs last year, down 7.3%. Much of that drop was due to the cut backs at Blockbuster, as well as the snafus at Netflix? DVD rental business. With that largely behind us, and rental kiosks becoming more popular, disc rentals will increase slightly to $6B in 2016. IHS says that kiosks accounted for 34% of disc rental spending last year, and will grow to 41% this year, and hit 52% in 2015. The silver lining for studios is that the online business is growing. ?If revenue were to be added from other viewing options such as video-on-demand, Internet-based sales and rentals, and subscription streaming from providers like Netflix and Hulu Plus?consumer spending across all outlets of home video would amount to nearly $17.2B,? says IHS senior analyst for U.S. video Michael Arrington. In a separate report, IHS said that spending for online movies more than doubled to $992M last year, mostly due to the growth of Netflix?s streaming service ? which passed Apple to be the No. 1 online movie provider. Web spending is expected to double again this year but then slow, the company says, unless a company such as HBO offers a standalone streaming service or someone offers a Web-based replacement for pay TV.
One of the best parts about being a celebrity is all the free stuff they get when they hit up splashy award shows, like the MTV Movie Awards taking place Sunday (June 3). And make no mistake, this year's Movie Awards gift bag is pretty impressive. It even includes one very fancy European getaway! Among [...]
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Protein pathways provide clues in leukemia researchPublic release date: 31-May-2012 [ | E-mail | Share ]
Contact: David Ruth david@rice.edu 713-348-6327 Rice University
Rice, MD Anderson scientists hope detailing networks will help fine-tune therapies
Scientists at Rice University and the University of Texas MD Anderson Cancer Center have successfully profiled protein pathways found to be distinctive to leukemia patients with particular variants of the disease.
Their research involved the creation of a new computational approach to identifying complex networks in protein signaling. They reported their work this month in the journal Proteomics.
With their new method, Rice bioengineer Amina Qutub, MD Anderson clinician and scientist Steven Kornblau and Rice graduate student Heather York analyzed more than a decade's worth of data. They identified patterns in the expression of 203 proteins studied in cells of acute myeloid leukemia (AML) patients and found nearly 700 protein pairs (from among thousands of possibilities) implicated in such networks.
The goal is to fine-tune therapy for patients, Qutub said. If it's possible to know the protein signaling pathways of particular types of leukemia, she said, then drugs may be found or designed to target the disease by blocking those pathways.
"Leukemia is not just one disease," said Qutub, who attended Rice as an undergraduate and is now an assistant professor of bioengineering based at Rice's BioScience Research Collaborative. "It's many, many diseases lumped into one category. You can't use a sledgehammer approach to treat it, though that's what clinicians have been doing."
"A Phillips head screwdriver does great if you've got a cross on your screw head," Kornblau said. "It doesn't work very well if you've got a slot. Companies have drugs that might work gangbusters in a small fraction of patients, but if you study a large aggregate, it doesn't look like a very active agent. Ideally, if it works in 5 percent of patients and you can pick them out ahead of time, you put them on the right drug and spare the other 95 percent from getting a drug that is not going to help them."
It's not unusual for leukemia patients to go into remission, Qutub said, but most suffer a relapse within two years. "We're trying to figure out what's so different about these patients, and whether we can enhance their therapy. The only way we know to do that is to figure out the protein signature."
Proteins are the workhorses of the cell. Created from blueprints stored in DNA, they pass chemical signals from one to another with instructions on just about every activity a cell is capable of. But when their basic coding is scrambled, they can just as easily cause a cell to self-destruct.
Qutub likened protein signaling pathways to city traffic. "You might take a train, drive a car, walk or take a bicycle, but eventually you would get to your location," she said. "In cancer patients, protein pathways seem to follow patterns. In normal cells, there are predominant pathways, and while you may have the same pathways in cancer cells, they might also have alternate routes and take them, to varying degrees."
Specific routes proteins use to pass signals may be the key to determining their type, so it was worth investigating a treasure trove of data that had been gathered from MD Anderson patients, Qutub said.
The project began with a chance meeting by Qutub and Kornblau at a conference in Colorado on hypoxia. "Steve said, 'You're working with mathematical modeling of hypoxic response. I have a patient set I think you might be interested in.' That's how it started, and it took us down a very different road, using machine-learning algorithms and statistical techniques that hadn't been part of my core research but have now become integral to both this project and other research in the lab," Qutub said.
The World Health Organization had categorized AML patients by certain cell phenotypes, Qutub said, "but very little had been done at the protein level. Our clinical collaborators had started genetic and proteomic studies a while back and identified recurrent patterns of protein expression, some of which were associated with recurrent chromosomal changes like 'inv (16),' the inversion of chromosome 16, or 't(8;21),' which is the transpose of the 8 and 21 chromosomes." They are found in about 15 percent of patients with AML, she said.
"Patients with these two chromosomal changes respond more favorably to current AML therapy," Kornblau said, "and the genes from each of these genetic abnormalities are involved in the same transcription factor complex, creating a mechanistic link. But despite this mechanistic link, they differ in some clinical features and outcome."
Kornblau's data was precisely what Qutub and her group needed. They targeted inv (16) and t(8;21) to learn what protein pathways were common to both of these cytogenetic categories, what might explain their increased responsiveness and what changes were unique to each that might explain their clinical differences. From the mass of data more than 20,000 possible protein pairs they found that 678 pairs related to inv (16) and t(8;21) were significantly different compared with normal cells.
"There are some genetic markers that are definitely associated with a prognosis of leukemia," Qutub said. "And other things can cause leukemia -- acute myeloid in particular -- perhaps even chemotherapy itself. The way cancer patients are treated may be causing mutations and in turn triggering leukemic response."
With their analytical method tried and tested, Qutub and her team can now statistically group patients who present with particular protein pathways by age, gender, therapies and other criteria, and then take a closer look at protein signaling pathways in bone marrow-derived stem cells, a prime suspect in patients who relapse even after decades of remission. "We think something has to be unique about stem cells," she said.
The key problem with all cancers, including leukemia, is their plasticity, she said. "Cancer has multiple ways of getting to the same destination. You try one drug, and cancer's going to circumvent you and go a different way. What we're trying to say is, 'Here are all the possible ways,' so we can begin to target the intersections."
###
The research was supported by the Leukemia and Lymphoma Society and Rice University.
Related links:
Read the abstract at http://onlinelibrary.wiley.com/doi/10.1002/pmic.201100491/abstract
Qutub Lab: http://www.qutublab.rice.edu/
BioScience Research Collaborative: http://brc.rice.edu/home/
New research by scientists at Rice University and the University of Texas MD Anderson Cancer Center profiles protein pathways distinctive to certain leukemia patients. The graphic shows interacting proteins in leukemia cells that can be characterized by networks analogous to highways. (Qutub Lab/Rice University)
Located on a 300-acre forested campus in Houston, Rice University is consistently ranked among the nation's top 20 universities by U.S. News & World Report. Rice has highly respected schools of Architecture, Business, Continuing Studies, Engineering, Humanities, Music, Natural Sciences and Social Sciences and is known for its "unconventional wisdom." With 3,708 undergraduates and 2,374 graduate students, Rice's undergraduate student-to-faculty ratio is 6-to-1. Its residential college system builds close-knit communities and lifelong friendships, just one reason why Rice has been ranked No. 1 for best quality of life multiple times by the Princeton Review and No. 4 for "best value" among private universities by Kiplinger's Personal Finance. To read "What they're saying about Rice," go to www.rice.edu/nationalmedia/Rice.pdf.
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AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.
Protein pathways provide clues in leukemia researchPublic release date: 31-May-2012 [ | E-mail | Share ]
Contact: David Ruth david@rice.edu 713-348-6327 Rice University
Rice, MD Anderson scientists hope detailing networks will help fine-tune therapies
Scientists at Rice University and the University of Texas MD Anderson Cancer Center have successfully profiled protein pathways found to be distinctive to leukemia patients with particular variants of the disease.
Their research involved the creation of a new computational approach to identifying complex networks in protein signaling. They reported their work this month in the journal Proteomics.
With their new method, Rice bioengineer Amina Qutub, MD Anderson clinician and scientist Steven Kornblau and Rice graduate student Heather York analyzed more than a decade's worth of data. They identified patterns in the expression of 203 proteins studied in cells of acute myeloid leukemia (AML) patients and found nearly 700 protein pairs (from among thousands of possibilities) implicated in such networks.
The goal is to fine-tune therapy for patients, Qutub said. If it's possible to know the protein signaling pathways of particular types of leukemia, she said, then drugs may be found or designed to target the disease by blocking those pathways.
"Leukemia is not just one disease," said Qutub, who attended Rice as an undergraduate and is now an assistant professor of bioengineering based at Rice's BioScience Research Collaborative. "It's many, many diseases lumped into one category. You can't use a sledgehammer approach to treat it, though that's what clinicians have been doing."
"A Phillips head screwdriver does great if you've got a cross on your screw head," Kornblau said. "It doesn't work very well if you've got a slot. Companies have drugs that might work gangbusters in a small fraction of patients, but if you study a large aggregate, it doesn't look like a very active agent. Ideally, if it works in 5 percent of patients and you can pick them out ahead of time, you put them on the right drug and spare the other 95 percent from getting a drug that is not going to help them."
It's not unusual for leukemia patients to go into remission, Qutub said, but most suffer a relapse within two years. "We're trying to figure out what's so different about these patients, and whether we can enhance their therapy. The only way we know to do that is to figure out the protein signature."
Proteins are the workhorses of the cell. Created from blueprints stored in DNA, they pass chemical signals from one to another with instructions on just about every activity a cell is capable of. But when their basic coding is scrambled, they can just as easily cause a cell to self-destruct.
Qutub likened protein signaling pathways to city traffic. "You might take a train, drive a car, walk or take a bicycle, but eventually you would get to your location," she said. "In cancer patients, protein pathways seem to follow patterns. In normal cells, there are predominant pathways, and while you may have the same pathways in cancer cells, they might also have alternate routes and take them, to varying degrees."
Specific routes proteins use to pass signals may be the key to determining their type, so it was worth investigating a treasure trove of data that had been gathered from MD Anderson patients, Qutub said.
The project began with a chance meeting by Qutub and Kornblau at a conference in Colorado on hypoxia. "Steve said, 'You're working with mathematical modeling of hypoxic response. I have a patient set I think you might be interested in.' That's how it started, and it took us down a very different road, using machine-learning algorithms and statistical techniques that hadn't been part of my core research but have now become integral to both this project and other research in the lab," Qutub said.
The World Health Organization had categorized AML patients by certain cell phenotypes, Qutub said, "but very little had been done at the protein level. Our clinical collaborators had started genetic and proteomic studies a while back and identified recurrent patterns of protein expression, some of which were associated with recurrent chromosomal changes like 'inv (16),' the inversion of chromosome 16, or 't(8;21),' which is the transpose of the 8 and 21 chromosomes." They are found in about 15 percent of patients with AML, she said.
"Patients with these two chromosomal changes respond more favorably to current AML therapy," Kornblau said, "and the genes from each of these genetic abnormalities are involved in the same transcription factor complex, creating a mechanistic link. But despite this mechanistic link, they differ in some clinical features and outcome."
Kornblau's data was precisely what Qutub and her group needed. They targeted inv (16) and t(8;21) to learn what protein pathways were common to both of these cytogenetic categories, what might explain their increased responsiveness and what changes were unique to each that might explain their clinical differences. From the mass of data more than 20,000 possible protein pairs they found that 678 pairs related to inv (16) and t(8;21) were significantly different compared with normal cells.
"There are some genetic markers that are definitely associated with a prognosis of leukemia," Qutub said. "And other things can cause leukemia -- acute myeloid in particular -- perhaps even chemotherapy itself. The way cancer patients are treated may be causing mutations and in turn triggering leukemic response."
With their analytical method tried and tested, Qutub and her team can now statistically group patients who present with particular protein pathways by age, gender, therapies and other criteria, and then take a closer look at protein signaling pathways in bone marrow-derived stem cells, a prime suspect in patients who relapse even after decades of remission. "We think something has to be unique about stem cells," she said.
The key problem with all cancers, including leukemia, is their plasticity, she said. "Cancer has multiple ways of getting to the same destination. You try one drug, and cancer's going to circumvent you and go a different way. What we're trying to say is, 'Here are all the possible ways,' so we can begin to target the intersections."
###
The research was supported by the Leukemia and Lymphoma Society and Rice University.
Related links:
Read the abstract at http://onlinelibrary.wiley.com/doi/10.1002/pmic.201100491/abstract
Qutub Lab: http://www.qutublab.rice.edu/
BioScience Research Collaborative: http://brc.rice.edu/home/
New research by scientists at Rice University and the University of Texas MD Anderson Cancer Center profiles protein pathways distinctive to certain leukemia patients. The graphic shows interacting proteins in leukemia cells that can be characterized by networks analogous to highways. (Qutub Lab/Rice University)
Located on a 300-acre forested campus in Houston, Rice University is consistently ranked among the nation's top 20 universities by U.S. News & World Report. Rice has highly respected schools of Architecture, Business, Continuing Studies, Engineering, Humanities, Music, Natural Sciences and Social Sciences and is known for its "unconventional wisdom." With 3,708 undergraduates and 2,374 graduate students, Rice's undergraduate student-to-faculty ratio is 6-to-1. Its residential college system builds close-knit communities and lifelong friendships, just one reason why Rice has been ranked No. 1 for best quality of life multiple times by the Princeton Review and No. 4 for "best value" among private universities by Kiplinger's Personal Finance. To read "What they're saying about Rice," go to www.rice.edu/nationalmedia/Rice.pdf.
[ | E-mail | Share ]
?
AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.
Mark Pitt, a fellow IT Pro member, contacted me last week and let me know that he enjoyed the IT Pro site. Thanks, Mark, for the encouragement! Mark works for an organization called the Information Security and Business Continuity Academy (IS & BCA), which studies business and security standards. Specifically, they show companies how to implement ISO standards, which are critical to business continuity and security.
Mark sent me a useful link to a summary of a standard called ISO 22301, the latest effort to outline procedures and practices that are designed to make sure businesses, well, stay in business in spite of various disasters. The new ISO 22301 standard replaces the older BS 25999-2 standard. The new standard includes new terms and procedures designed to help businesses plan for various eventualities. According to the link Mark sent me, the summary compares the new ISO 22301 standard to the older standard. New terms found in ISO 22301 include:
Disruptive incident
Maximum acceptable outage
Minimum business continuity objective
Maximum data loss
I wanted to share this link with you, because it explain the significant changes that ISO has made. Like most people, I've found ISO-based discussions to be, well, really arcane. But trust me: If your job has anything to do
with any type of server or important IT-related service, this standard is relevant to you. I mean it. Your work will be impacted by this new standard, even if you don't quite realize it. Let me
put it another way: You're already using ISO stuff, though few workers
quite realize that they are.
Let me tell you a story about how, well, difficult ISO standards can be to read and understand, at least at first. Years ago, I wanted to purchase an ISO standard to help me create a security course. Back then, trying to purchase a standard from the ISO Web site practically required learning some sort of ritualistic, pan-European handshake. You should have seen the hoops I had to jump through just to get a copy of the document. Just obtaining the document was a painful process. And, I have to say, reading through one of these documents was about as bad as reading a bad Victorian novel: Long, dry, and full of all sorts of (seemingly) irrelevant information.
But slowly, I realized that ISO standards contribute many important things to us. First of all, they codify important procedures. They also give everyone - IT workers, managers and vendors alike - a common language to use. So, I strongly recommend that you familiarize yourself with the summary Mark provided. While I doubt that quoting from an ISO standard will gain you many friends in the IT world, understanding the concepts found in the document and then learning more about them will turn you into an expert. That's always worthwhile.
KITTANNING, Pa. (AP) ? State police say a pair of amorous bovines tied up traffic near the intersection of two rural Pennsylvania highways.
The Pittsburgh Post-Gazette first reported the situation Friday morning. Trooper John Corna tells The Associated Press it was resolved about 9:15 a.m. when state Department of Agriculture managed to shoo the bull and cow into a trailer.
Corna says the intersection of Routes 28 and 85 in Rayburn Township is busier than usual because of construction, and traffic was hampered when the mating bovines refused to budge. The intersection is about 40 miles northeast of Pittsburgh.
Corna says troopers "kept trying to shoo them off the highway, but that just got the bull mad and it started to escalate."
The animals are at a nearby farm until their owner is identified.
Detroit Red Wings captain Nicklas Lidstrom of Sweden announces his retirement during a news conference in Detroit, Thursday, May 31, 2012. Lidstrom retires after a 20-season career. (AP Photo/Carlos Osorio)
Detroit Red Wings captain Nicklas Lidstrom of Sweden announces his retirement during a news conference in Detroit, Thursday, May 31, 2012. Lidstrom retires after a 20-season career. (AP Photo/Carlos Osorio)
Detroit Red Wings captain Nicklas Lidstrom of Sweden hugs team owner Marian Ilitch, as his wife, Annika, and eight-year-old son, Lucas, look on, after announcing his retirement during a news conference in Detroit, Thursday, May 31, 2012. Lidstrom retires after a 20-season career. (AP Photo/Carlos Osorio)
Detroit Red Wings captain Nicklas Lidstrom of Sweden and his wife Annika arrive for the news conference where he announced his retirement in Detroit, Thursday, May 31, 2012. Lidstrom retires after a 20-season career. (AP Photo/Carlos Osorio)
Detroit Red Wings owners Mike and Marian Ilitch, foreground, are followed by team captain Nicklas Lidstrom of Sweden and his wife Annika, as they arrive for the news conference where Lidstrom announced his retirement, in Detroit, Thursday, May 31, 2012. Lidstrom retires after a 20-season career. (AP Photo/Carlos Osorio)
Detroit Red Wings captain Nicklas Lidstrom of Sweden is interviewed after announcing his retirement in Detroit, Thursday, May 31, 2012. Lidstrom retires after a 20-season career. (AP Photo/Carlos Osorio)
DETROIT (AP) ? Nicklas Lidstrom is so used to being great that the star defenseman refused to settle for just being good.
Lidstrom retired after 20 quietly spectacular seasons with the Detroit Red Wings, leaving a legacy of greatness on and off the ice along with perhaps another $6 million he could have made with a third straight one-year contract.
The four-time Stanley Cup champion and seven-time Norris Trophy winner fought back tears as he made the announcement Thursday. He said he knew it was time to end one of the best careers in NHL history when he started to work out recently.
"My drive and motivation are not where they to need to be to play at this level," Lidstrom said.
The 42-year-old Swede set an NHL record by playing 1,564 games with a single team. He had put retirement on hold in each of the previous two years by signing one-year contracts.
"I've been dreading this day since I became manager in 1997," Red Wings general manager Ken Holland said.
When Lidstrom told him last week that he was retiring, Holland said he could have the weekend, weeks or even months to think about it more in the hopes that he would change his mind. Holland now has $20-plus million in salary cap space to attempt to sign a standout defenseman, perhaps Nashville's Ryan Suter if the Predators can't re-sign him before July.
San Jose Sharks general manager Doug Wilson, though, said what everyone in the Motor City is thinking.
"You don't replace players like that," Wilson said.
No, you don't.
Even when Lidstrom didn't have one of his best years, such as last season, he was still the storied franchise's best player on the blue line and one of the better defensemen in the league.
Lidstrom had 34 points and a plus-21 rating that ranked among the league leaders, and for his career wound up with 264 goals and 1,142 points. After being incredibly durable for 19 seasons, he missed a career-high 11 games last season with a bruised right ankle and was out for another game with the flu.
"That didn't sway me one way or another," Lidstrom said. "A couple weeks after the season is over, you start working out. Once I started doing that, I didn't have the push I need, and I can't cheat myself."
He plans to move his family to Sweden and hopes to have an off-ice role with the Red Wings.
"Retiring today allows me to walk away with pride, rather than have the game walk away from me," said Lidstrom, whose oldest of four sons went to Sweden two years ago to attend school and play hockey.
Lidstrom's wife, Annika, left the door open for her husband to keep playing even though that in effect would often make her a single parent for another season.
"She even said, 'If you want to play another year, we can make this work," he recalled in one of many emotional moments at Joe Louis Arena.
Lidstrom was named the NHL's best defenseman last year for a seventh time since 2001, matching Doug Harvey's total and trailing Bobby Orr's league record by one. When Lidstrom won his final Norris Trophy last summer, he was a finalist for the 11th time in 13 seasons.
Defenseman Brad Stuart, who was his teammate the past four-plus seasons, said he was amazed at Lidstrom's ability to make the right play on almost every shift game after game.
"I've played with great players who made mistakes, but I can't think of one game when I thought, 'Nick just didn't have it tonight,'" Stuart said during this year's one-series postseason. "He's that same, steady, amazing defenseman every night. I think I've seen him out of breath maybe three or four times in a few years because he's so smart, he gets himself in the right position to make a play."
The four-time Olympian scored the gold-medal winning goal for Sweden over Finland in 2006. He became the first European-born captain to win a Stanley Cup in 2008, six years after being the first from Europe to win the Conn Smythe Trophy as playoffs MVP.
New Jersey Devils defenseman Henrik Tallinder, a fellow Swede, said Lidstrom is an icon at home.
"In my eyes, he's the best Swedish player we've had over here," Tallinder said. "No offense to (Peter) Forsberg and (Mats) Sundin. Just with four Stanley Cups, seven Norris Trophies, that says it all, I think.
Lidstrom's 6-foot-2, 190-pound body is chiseled thanks to a year-round workout that includes exercise before practice and after games along with a sensible diet that includes only occasional slices of pizza and fast food. His teammates called him "The Perfect Human," in part because he's as humble as he is successful on the ice.
As he said goodbye, Lidstrom thanked the owners, front office staff, coaches and teammates ? as all retiring players do ? and then added his own touch by praising behind-the-scenes contributors such as Leslie Baker, who serves meals to players and their families.
"It's one of the most emotional days in Red Wings history with Nick retiring and all you people showing your respect for such a high-quality individual," Red Wings owner Mike Ilitch said at the packed news conference that included team employees wearing Lidstrom's No. 5 red jersey with a winged wheel.
When Ilitch's wife, Marian, spoke about Lidstrom after the news conference, tears rolled down both of her cheeks.
"I'm happy for him and his family ? as sad as I am to lose him," she said. "It's a bittersweet day."
Steve Yzerman's No. 19 jersey became the sixth retired by the storied franchise and was hoisted to the rafters in 2007 alongside Gordie Howe's No. 9, Ted Lindsay's No. 7, Terry Sawchuck's No. 1, Alex Delvecchio's No. 10 and Sid Abel's No. 12.
Lidstrom's No. 5 will likely be next.
"I think he's been the most valuable player of his era and will go down as one of the greatest Red Wings of all time," Holland said.
___
AP Sports Writers Ira Podell in New York and Tom Canavan in Newark, N.J., contributed to this report.
___
Follow Larry Lage on Twitter at www.Twitter.com/larrylage
Spending for DVDs and Blu-ray discs is falling so fast that 2012 likely will be first year when consumers will pay more to buy and rent movies via the Internet, according to a report today from IHS Screen Digest Video Intelligence Service. The research firm says that the average household will spend $123.50 to buy and rent discs this year ? down from $133.21 last year and $149.53 in 2010. The average home spent $206.78 in 2006, when Blu-ray discs were introduced. Looked at another way, people spent $8.8B last year to buy packaged videos, down 12% vs 2010. That will keep falling to $5.4B in 2016 ? which IHS notes is ?the level in 1997 when DVDs were first launched.? Meanwhile, consumers paid $5.7B to rent discs last year, down 7.3%. Much of that drop was due to the cut backs at Blockbuster, as well as the snafus at Netflix? DVD rental business. With that largely behind us, and rental kiosks becoming more popular, disc rentals will increase slightly to $6B in 2016. IHS says that kiosks accounted for 34% of disc rental spending last year, and will grow to 41% this year, and hit 52% in 2015. The silver lining for studios is that the online business is growing. ?If revenue were to be added from other viewing options such as video-on-demand, Internet-based sales and rentals, and subscription streaming from providers like Netflix and Hulu Plus?consumer spending across all outlets of home video would amount to nearly $17.2B,? says IHS senior analyst for U.S. video Michael Arrington. In a separate report, IHS said that spending for online movies more than doubled to $992M last year, mostly due to the growth of Netflix?s streaming service ? which passed Apple to be the No. 1 online movie provider. Web spending is expected to double again this year but then slow, the company says, unless a company such as HBO offers a standalone streaming service or someone offers a Web-based replacement for pay TV.
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Public release date: 31-May-2012
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